منابع مشابه
Ischaemic heart disease.
There can be no truly objective frequency estimate of the susceptibility of the population to ischaemic heart disease: everybody has some degree of susceptibility. The disease kills 1 in 4 of the population, and 1 man in 10 dies of ischaemic heart disease before 65 years of age. The major burden is mortality, though there is often some degree of impairment among survivors of acute attacks. It i...
متن کاملMetabolomics and ischaemic heart disease.
Ischaemic heart disease accounts for nearly half of the global cardiovascular disease burden. Aetiologies relating to heart disease are complex, but dyslipidaemia, oxidative stress and inflammation are cardinal features. Despite preventative measures and advancements in treatment regimens with lipid-lowering agents, the high prevalence of heart disease and the residual risk of recurrent events ...
متن کاملNon-atheromatous ischaemic heart disease.
Myocardial ischaemia may result from congenital abnormalities in the origin or distribution of the coronary arteries. Coronary obstruction may also develop at a young age in some rare hereditary disorders such as Hurler's syndrome, homocystinuria or pseudoxanthoma elasticum. The coronary arteries may be the site of embolism; ostial stenosis may complicate luetic (or other) aortitis or coronary ...
متن کاملThrombosis and ischaemic heart disease.
The overwhelming dominance of the lipid hypothesis for atheroma has obscured the compelling evidence-some of it of many years standing' but some that is more recent2 3 -that both platelets and the coagulation system also contribute to the lesion. There is much epidemiological evidence4 5 of some other process besides atherogenesis to account for the clinical manifestations of coronary artery di...
متن کاملAetiology of ischaemic heart disease.
There is evidence from population studies of strong positive associations between smoking, arterial pressure and serum cholesterol concentration and risk of ischaemic heart disease, and these appear to be independent of one another, but the evidence from unifactorial trials of risk factor reduction is less impressive. For smoking, there is no good experimental evidence that cessation reduces th...
متن کاملذخیره در منابع من
با ذخیره ی این منبع در منابع من، دسترسی به آن را برای استفاده های بعدی آسان تر کنید
ژورنال
عنوان ژورنال: Journal of Nepal Medical Association
سال: 1970
ISSN: 1815-672X,0028-2715
DOI: 10.31729/jnma.1018